57 years since the assassination of President John F. Kennedy

first_imgKennedy was shot on November 22, 1963 in Dallas, Texas as his motorcade drove through the city. His presidency included a number of foreign affairs accomplishments, as well as tensions. JFK was the president during the Cold War. Kennedy was laid to rest in Arlington National Cemetary just three days later. JFK was the 35th President of the United States and one of the youngest. JFK’s legacy includes the creation of the Peace Corps, sending the Civil Rights Act bill to Congress, and the famed lines in his 1961 inauguration speech, “Ask not what your country can do for you, but what you can do for your country.” Kennedy was struck twice in the neck and head. Lee Harvey Oswald was arrested for the assassination, but he was shot and killed before going to trial. (WBNG) — Sunday marks the 57 year anniversary of the assassination of President John F. Kennedy. last_img read more

No ‘crow’ for woman caught speeding to help injured bird

first_imgA woman was caught speeding after rushing to help a bird that was in distress.Tara Lafferty works for a Co Donegal animal charity and was answering a call in Letterkenny on July 20th last. Ms Lafferty appeared at Letterkenny District Court today charged with speeding at 62kph in a 50kph zone at Ballyraine, Letterkenny.Ms Lafferty, of Kilbarron, Rossnowlagh told the court “I work for an animal charity and I was on my way to inject a crow when this happened.”She said she did not pay the speeding fine as she wanted to explain to the court what had happened.She asked Judge Paul Kelly if he could avoid giving her penalty points on her license.He replied that it was out of his hands but reduced the fine to €50 in the circumstances.No ‘crow’ for woman caught speeding to help injured bird was last modified: October 1st, 2018 by StephenShare this:Click to share on Facebook (Opens in new window)Click to share on Twitter (Opens in new window)Click to share on LinkedIn (Opens in new window)Click to share on Reddit (Opens in new window)Click to share on Pocket (Opens in new window)Click to share on Telegram (Opens in new window)Click to share on WhatsApp (Opens in new window)Click to share on Skype (Opens in new window)Click to print (Opens in new window)last_img read more

Study shows how the drive to eat overpowers the brains signal to

first_imgBy Sally Robertson, B.Sc.Sep 21 2018University of Michigan researchers have studied two groups of brain cells that compete for the control of feeding behaviour and found that cells that drive the urge to eat overpower the ones that signal to stop. Source:https://www.eurekalert.org/pub_releases/2018-09/mm-u-fbs092018.php Lightspring | ShutterstockThe study also showed that the brain’s opioid system is involved and that administering a drug called naloxone can block this system.They say their findings could help to inform the fight against the global obesity epidemic.In a mouse model, Huda Akil and colleagues studied two groups of brain cells called pro-opiomelanocortin (POMC) and AgRP (Agouti-Gene-Related Peptide). The two groups are located near to each other within a region of the brain called the arcuate nucleus, which is part of the behavior-regulating hypothalamus.Previous studies have demonstrated that POMC responds to certain signals in the body by restricting the urge to eat, while AgRP drives the urge to eat, particularly when food is scarce or when a long time has passed since last eating.As reported in the Proceedings of the National Academy of Science, the current study has now shown how the two groups of cells relate to each other. Using a technique called optogenetics, Akil and team stimulated POMC, given that POMC neurons had previously been shown to play a role in feeding behaviour.However, the result of stimulating the POMC cells was that a group of neighboring AgRP cells were also stimulated. These two groups of cells are derived from the same parent cells during embryonic development, meaning that the technique the researchers used to target POMS also captured the AgRP nerve cells.Related StoriesRush University Medical Center offers new FDA-approved treatment for brain aneurysmsRepurposing a heart drug could increase survival rate of children with ependymomaWearing a hearing aid may mitigate dementia riskInterestingly, the team found that when both groups of cells were activated, it was the “keep eating” signal from AgRP that had the most influence on eating behavior. That AgRP signal was more powerful than the “stop eating” signal generated by POMC.”When both are stimulated at once, AgRP steals the show,” says Akil.The researchers then used a technique called c-fos activation to further investigate the downstream effects of activating POMC and AgRP.They found that activating AGRP also activated the brain’s opioid system and that administering the opioid antagonist drug naloxone stopped the urge to eat.“This suggests that the brain’s own endogenous opioid system may play a role in wanting to eat beyond what is needed,”Akil.The finding led Akil and team to wonder whether bombardment of the senses with the things we see, smell and socially interact with relating to food may be involved with the urge to overeat.She thinks perhaps these factors combine to trigger us to become interested in eating when we are not even hungry: “There’s a whole industry built on enticing you to eat, whether you need it or not, through visual cues, packaging, smells, emotional associations. People get hungry just looking at them, and we need to study the neural signals involved in those attentional, perceptional mechanisms that drive us to eat.”last_img read more